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Caffeine and Autophagy To Reduce Fats in Liver

Duration: 30:28

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william_j_upton@*.com

Jan 17 2024, 8:21 pm

I guess I need to add more caffine to my diet to fight my diabetes’ tastings has already removed my imflamation

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fisher6000@*.com

Jan 30 2023, 11:16 am

I follow your Youtube videos, Podcasts and lectures. I appreciate all your hard work put into FLCCC content, Spike Protein Diseases, and Autophagy. Your cartoon talents are a huge plus in helping to understand lecture content. Thank You Dr. Been for your consistently amazing work.

Caffeine and Autophagy To Reduce Fats in Liver

This study from Singapore demonstrates how Caffeine helps reduce lipid fats by autophagy. Let's review.

Disclaimer:
This video is not intended to provide assessment, diagnosis, treatment, or medical advice; it also does not constitute provision of healthcare services. The content provided in this video is for informational and educational purposes only.
Please consult with a physician or healthcare professional regarding any medical or mental health related diagnosis or treatment. No information in this video should ever be considered as a substitute for advice from a healthcare professional.

URL list from Thursday, Aug. 4 2022

Caffeine stimulates hepatic lipid metabolism by the autophagy‐lysosomal pathway in mice - Sinha - 2014 - Hepatology - Wiley Online Library
https://aasldpubs.onlinelibrary.wiley.com/doi/10.1002/hep.26667

Impact of coffee on liver diseases: a systematic review - Saab - 2014 - Liver International - Wiley Online Library
https://onlinelibrary.wiley.com/doi/10.1111/liv.12304

Coffee: The magical bean for liver diseases - PMC
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5440772/

Oleic acid - Wikipedia
https://en.wikipedia.org/wiki/Oleic_acid

Lipid peroxidation - Wikipedia
https://en.wikipedia.org/wiki/Lipid_peroxidation

Lipid Oxidation - an overview | ScienceDirect Topics
https://www.sciencedirect.com/topics/medicine-and-dentistry/lipid-oxidation

Sphingolipids general structures - Sphingolipid - Wikipedia
https://en.wikipedia.org/wiki/Sphingolipid#/media/File:Sphingolipids_general_structures.png

The Emerging Roles of mTORC1 in Macromanaging Autophagy - PMC
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6826502/

ATG5 and ATG7 induced autophagy interplays with UPR via PERK signaling | Cell Communication and Signaling | Full Text
https://biosignaling.biomedcentral.com/articles/10.1186/s12964-019-0353-3

The Beclin 1 network regulates autophagy and apoptosis - PMC
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3131912/

* Recognize caffeine's role in reducing liver lipid content.

* Understand the genetic, pharmacological, and metabolomic methods employed in in vitro and in vivo studies.

* Grasp how caffeine triggers raw material formation for phagosomes and lysosomes.

* Identify the impact of caffeine on mTOR, amino acids, sphingolipids, and genetic expression.

* Recognize caffeine's protective effect on mitochondria and its influence on lysosomal acidification.

Following answers are created by ChatGPT. Occasionally the answer may be harmful, incorrect, false, misleading, incomplete, or limited in knowledge of world. Please contact your doctor for all healthcare decisions. Also, double check the answer provided by the AI below.

Faculty

In addition to the presenter, following authors may have helped with the content writing, review, or approval:

  • Dr. Mobeen Syed

CME, CE, CEU and Other Credit Types:

ACCME Accreditation Statement
The DrBeen Corp is accredited by the Accreditation Council for Continuing Medical Education (ACCME) to provide continuing medical education for physicians.

AMA Credit Designation Statement
The DrBeen Corp designates this enduring material for a maximum of 0.5 AMA PRA Category 1 Credits™. Physicians should claim only the credit commensurate with the extent of their participation in the activity.


Disclosure Information

In accordance with the disclosure policies of DrBeen Corp and the ACCME (Accreditation Council for Continuing Medical Education), we are committed to upholding principles of balance, independence, objectivity, and scientific rigor in all of our Continuing Medical Education (CME) and Continuing Education (CE) activities. These policies include the careful management and mitigation of any relevant financial relationships with organizations that are not eligible.
All members of the Activity Planning Committee and presenters have disclosed their relevant financial relationships. The DrBeen Corp CE Committee has thoroughly reviewed these disclosures and determined that these relationships are not deemed inappropriate in the context of their respective presentations. Additionally, they are found to be consistent with the educational objectives and the integrity of the activity.

Faculty Disclosures
Dr. Mobeen Syed Author declares no conflict of interest.

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